In the U.S., if mostly solid or tumorlike lesion represented the most important differential diagnoses to be considered
following primary and secondary liver tumors. Patients with such injuries, more research, imaging such as CT
or magnetic resonance imaging and biopsy, aspiration or defeat for cytological or histological examination can be performed. In some cases, other more invasive procedures or treatment, such as small or large hepatectomy were performed (
). In our study we showed that predominantly solid U.S. appearance may occur with monomicrobial
liver abscesses to pneumonia, with 94% of them predominantly solid liver abscesses associated with the
to pneumonia. More than half of patients with these abscesses had diabetes. Based on these data, we propose that the
for diabetics that Chinese or Asian ethnicity and that the presentation is sepsis, right upper quadrant
pain, abnormal liver function, and mostly solid defeat in the U.S. , also include a preliminary diagnosis
Go to liver abscess, pneumonia. In pneumonia liver abscess associated with a relatively small amount of pus in the initial aspiration. Our current standard treatment of purulent >> << hepatophyma more than 3 cm in diameter antibiotics and percutaneous drainage or repeated aspiration. In our study for polymicrobial or nonBЂ "
To pneumonia purulent abscesses monomicrobial liver, the initial desire to give more than 10 ml of pus in more than 80% of patients. In >> << On the other hand, for monomicrobial
liver abscesses to pneumonia, the initial commitment made at least 10 ml of pus in all patients and less than 2 ml of pus in more than
80%. This is a bad harvest in an effort, probably due to association
Go to pneumonia liver abscesses with aggregation of several small cells that do not communicate, which means that these abscesses may
represent immature forms of liver abscess with failure liquefaction (
). Recently, it was noted that more than 5 cm diameter, concomitant sepsis, the formation of intrahepatic gas, sharp
physiology and chronic disease evaluation III, and APACHE III, score above 40, and delays or inadequate drainage is essential
factors poor prognosis in the
to pneumonia purulent abscesses of liver (). In addition, diabetes mellitus, large abscess, gas formation in the liver abscess and left shares of are risk factors
spontaneous rupture in patients with
Go to pneumonia liver abscess (). We believe that further prospective studies to determine the best treatment strategy for patients with
Go to the liver abscess, pneumonia. Association of liver abscesses to pneumonia in these predominantly solid appearance at U.S., probably due to their refusal to liquefaction. This
It was noted that festering abscesses of the liver in a Chinese population of patients with diabetes are often associated with
Go to pneumonia (,
). It was also shown that a high degree of genetic similarity is present in more than 60% >> << to strains of pneumonia () and high prevalence of resistance to phagocytosis serotype capsule is present in K strains of pneumonia associated with these liver abscesses (). Recently, virulence gene
Wizard, which encodes the 43-kDa outer membrane protein has been shown that more widespread in invasive strains
Go to the pneumonia that cause primary liver abscess and metastatic septic complications (). Wild type
magician to pneumonia strains of mucoviscous Web polysaccharide, active in spreading neimunnoho human serum, resisted phagocytosis, and
caused liver microabscess and meningitis in mice. But
Mage B € 'mutants without ekzopolisaharydu Web became very sensitive and serum and phagocytosis were susceptible avirulentnyy >> << in mice. Thus, it is likely that diabetic patients are prone to dysfunction with phagocytosis (
BЂ "), the
Mage + K pneumonia strains of this additional factor of virulence will abscesses very difficult fahotsytuyut. This poor phagocytosis
prevent killing microbe pneumonia K and thus liquefied abscess. As for the results of our patients, the overall mortality rate in our series (18%) concordant strattera 10mg with previously reported
rate (5. 2% BЂ "41. 0%) (,
BЂ"). In some studies (,
),
to pneumonia, liver abscess reported to decrease mortality, whereas in the present series, as in patients with
Go to pneumonia and in patients with nonBЂ "To liver abscesses pneumonia with similar mortality. Further investigation, as
to the strains of pneumonia and host susceptibility in different populations will need to find different mortality
different population groups. Our study had limitations. First, the criteria for the diagnosis does not apply prospectively. In a prospective study of predominantly
solid or predominantly cystic pattern on the basis of classification, to confirm its usefulness in the diagnosis of liver abscesses caused by
Go to pneumonia. Second, the proportion of liver abscesses caused by
Go to pneumonia in our study population seems relatively high compared with that in Western countries. Third, we did not include
assessment interobserver agreement in the classification of liver abscesses. However, we believe that the classification >> << simple and easy to use. Fourth, only 69% of patients with purulent liver abscesses with known pathogens
passed in the U.S.. Other patients were evaluated with CT only. This is probably the result of individual clinicians >> << advantages and lead to any bias in the results. Finally, unlike the classic cystic liver abscesses caused by other organisms of purulent,
to liver abscesses, pneumonia, usually less liquid with a predominantly solid appearance in the U.S. and associated with a small >> << , the number of pus obtained at initial aspiration. .
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